In patients with pulmonary embolism, an increase in dead space leads to inefficient gas exchange and hypoxemia.

The architect filled the dead spaces with small reading nooks and indoor plants to make the home feel cozier.

When performing the experiment, the technician measured the anatomic dead space by making a cast of the airways.

During the ventilation-perfusion study, the clinician measured the alveolar dead space to assess how much air in the alveoli was not participating in gas exchange.

In early respiratory physiology texts, physiologic dead space referred to the volume of the conducting airways of the lung, a definition that became inadequate after alveolar dead space was described.

In patients with pulmonary embolism, the increase in parallel dead space contributed to worsened gas exchange.

During the pilot test, the team noticed airflow inefficiencies caused by parallel dead spaces within the ventilation manifold.

When measuring tidal volume, clinicians must account for anatomic dead spaces to avoid overestimating effective ventilation.

In the ventilated regions of the lung, alveolar dead spaces can increase after a pulmonary embolism.

In ventilated patients, increased physiologic dead spaces often correlate with worse outcomes.