During infection, the injured tissue secreted a chemokine that rapidly recruited neutrophils and monocytes to the inflamed site.

During inflammation, immune cells follow gradients of chemokines to reach the damaged tissue.

Under the microscope, the neutrophils swarmed chemokinetically toward the higher concentration of chemoattractants.

Researchers observed that chemokinetics increased the random motility of the bacterial population in response to the uniform chemical stimulus.

The chemokinetic response of neutrophils allowed them to disperse rapidly toward the bacterial metabolite gradient.

Researchers observed chemokinesis in cell cultures, noting increased random movement when exposed to specific attractants.

Although chemokinemia is often measured in clinical studies, the normal range varies between individuals.