Clinicians monitored CD4 counts to determine how well the patients' immune systems were recovering.

Researchers measured the decline in CD4s after starting the new antiviral therapy.

Mutations in CD40, a costimulatory protein on antigen-presenting cells required for activation by CD154 of T helper cells, can cause hyper-IgM syndrome type 3 and recurrent infections.

CD45 is essential for proper signaling during hematopoiesis.

During the experiment, elevated CD42 levels correlated with increased platelet adhesion and more rapid clot formation at sites of vascular injury.

Researchers examined how CD46, a membrane protein that acts as an inhibitory complement receptor and serves as an entry point for measles virus, human herpesvirus-6, and Neisseria, mediates pathogen entry and regulates complement activation on host cells.

Researchers found that CD43 expression is reduced in patients with Wiskott-Aldrich syndrome, affecting leukocyte adhesion and signaling.